Type-2 diabetes mellitus is a breakdown in blood
sugar regulation that can begin to develop slowly and insidiously
in adulthood, or earlier, in adolescence or even childhood. Obesity,
sedentary lifestyle, poor diet, and genetic predisposition can gradually
desensitize the body to the actions of insulin, the hormone that
transports glucose from the bloodstream into cells. Eventually this
malfunction may trigger a vicious cycle of imbalances that promotes
further obesity and metabolic imbalances.
Inability to maintain normal glucose control can
lead to many metabolic diseases, including obesity, heart disease,
hypertension, diabetes, chronic fatigue, accelerated aging, as well
as numerous mental and emotional disorders. Unfortunately, without
early and accurate preventive testing, the first "sign"
that surfaces may be a permanent complication from type-2 diabetes
such as nerve loss, impotence, or even blindness.
The following functional assessments can detect
early signs of glucose and insulin dysregulation that may precede
type-2 diabetes by many years. These tests are also important for
monitoring the effectiveness of treatments aimed at improving metabolic
health. For more information click on link below:
Type-2
Diabetes and Cardiovascular Assessment: Diabetes and
its related metabolic imbalances are linked to a higher risk of
cardiovascular disease.
Type-2
Diabetes and Oxidative Stress: The potential for type-2
diabetes to destroy tissue and damage physiological function may
be mediated to a large degree by excess free radicals.
Type-2
Diabetes and Male Hormone Assessment: As men age, declining
levels of testosterone may "trigger" metabolic imbalances
that promote insulin resistance and type-2 diabetes.
Type-2
Diabetes and Elemental Analysis: Levels of many nutrient
and toxic elements are associated with patterns of blood sugar regulation
and coronary heart disease risk.
Type-2
Diabetes and Female Hormone Assessment:
Imbalances of androgens such as testosterone and DHEA are believed
to fuel insulin resistance and central obesity in women.