Osteoporosis is the progressive thinning of the bones that takes place when new bone formation does not keep up with bone removal, leaving the bone progressively brittle. As bone is lost, the skeleton continues to have a normal composition, but it becomes porous, hyper-mineralized and more fragile, leading to spinal deformities (dowager's hump) and painful fractures of the wrist, hip and spine.

Osteoporosis is the most common bone disorder in America.1 More than 50% of healthy American women aged 30-40 are likely to develop vertebral fractures as they age due to osteoporosis.2 Unfortunately, treatment is only partially successful (at best) once progressive bone weakening has occurred. It is important to identify women and men who are currently losing bone at an accelerated rate so that effective treatment can begin when the therapeutic burden is prevention instead of reversal of bone loss.

The following Genova Diagnostics assessments provide crucial information to establish effective intervention strategies against osteoporosis:

Osteoporosis and Bone Resorption: If bone loss is detected early enough, interventions can arrest loss and protect against future osteopororis and bone fractures.

Osteoporosis and Female Hormones: Deficiencies of important female sex hormones have been clinically linked to increased incidence of osteoporosis.

Osteoporsis and Testosterone: Though osteoporosis chiefly strikes women, one out of every eight men will also fall victim to this disease, and testosterone may play a key role.

Osteoporosis and Adrenal Hormones: The hormones DHEA and cortisol, produced in the body's adrenal glands, have a powerful impact on bone density.

Osteoporosis and Melatonin: A potent antioxidant and modulator of the sleep wake cycle, melatonin actually helps regulate the metabolism of calcium.

References:
1 Riffee JM. Amer Pharm 1992;Vol NS32; 8:61-72.
2 Prior JC et al. N Eng J Med 1990;323:1221-7.

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